VETERINARY ANATOMIC PATHOLOGY PHASE II
Pathology
JKP - Phase II Veterinary Pathology
Sample Data
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 Cause? {{c1::1. Dirofilaria immitis2. porcine
dermatitis and nephropathy syndrome from porcine circovirus 2 infection }}Other causes of {{c2::immune mediated glomerulonephritis: Borrelia burgdorferi –
membranoproliferative glomerulonephritisLeishmania – mesangioproliferative GN,
MPGN, GSFamilial – Bernese mountain dog, britanny
spaniel (MPGN)Horses – MPGN with EIA, strep equi,
herpesSwine: porcine dermatitis and nephropathy
syndrome – feeder pigs w/ PCV2 -
systemic necrotizing vasculitis à
proliferative GN, fibrin in tuft, cresents, interstitial nephritis; Hereditary factor H deficiency
(inhibitor of complement) -MPGNRuminants -hereditary MPGN in Finnish
landrace sheep, medi-visna, BVD}}
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 Lesion? {{c1::Large
foam cells that contain lipid may be found in lobules of glomerular tufts
(source unknown)}}Predisposing factors? {{c1::Can
occur w/ diabetes mellitus, dyslipidemia or following trauma }}Sequela? {{c1::Can cause
aneurysmal dilation of capillary loops with mesangiolysis}} |
| Back Extra | eFigure 4-25 Glomerular lipid emboli in a severely proteinuric adult dog. A. H&E. B. Masson trichrome. C. Jones’ methenamine silver. Note the distortion of glomerular capillary loops by cleared spaces. Almost all glomeruli in the biopsy core were similarly affected. Figure4-43 Glomerularlipidosisina1-year-olddogwithmarked proteinuria. A. PAS stain. B. TEM of a glomerular capillary reveals multiple osmiophilic lipid droplets. Immune complexes are not present. (Courtesy G. Lees, F. Clubb.) |
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 Cause? {{c1::Dioctophyma renale adult worms live in renal pelvis (may also encyst in body cavity, uterus, mammary gland, bladder)}}Affected species? {{c1:: Mink, dogs, cats, pig, ox, horse - mostly fish-eating carnivors}}Intermediate host/s? {{c1::life cycle involves 2 aquatic intermediate hosts}} |
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